Does Vitamin-C stabilize plaque?

[Lucky1]

[ofonorow]

First of all, vitamin C and lysine are completely nontoxic nutrients required for life. The only issue is dosage. So all people have some vitamin C and lysine in their systems. IMO, the healthier people have more.

Pauling's associate Rath has written that vitamin C and lysine (as Lp(a) binding inhibitors) remove Lp(a) based plaques "molecule by molecule." After all these years, we have no concerns that plaques in very seriously ill people will rupture (versus what routinely happens during surgical procedures.) People invariable become better, unless they feel cured and stop the protocol.

[Lucky1]

Thank you for the response. You say it removes lp(a) based plaques which implies there can be other than Lp(a) based plaques. Is that common? I've been doing the vitamin c protocol for a couple of months now and I guess my concern is, what if it's not a Lp(a) based plaque ? I really need to know some test that I can establish a base line for the plaque and check latter to see if the protocol is working. Testing for Lp(a) could tell me if that's being reduced unless the test measurement includes what the lysine and proline have bound up, but that doesn't help if the plaques is not Lp(a) plaque. Do you know of any repeatable non-invasive lab test that would tell me that? I've had a calcium score but have read varied accounts as to how useful that is for the purpose I speak of.
Thanks again for responding!!

[ofonorow]

There is always debate in science, and there has not been much direct science as there should have been regarding an outright cure for cardiovascular diseased. A series of discoveries, starting with Biesegal and Matthias' Rath's team in Germany seemed to find Lp(a) (and only Lp(a) cholesterol) in atherosclerotic plaques. The Pauling/Rath theory is that the "sticky" Lp(a) acts as a surrogate for low vitamin C levels, "i.e., patching" arteries that would otherwise be strong from collagen if there were sufficient vitamin C. So I would say that all atherosclerotic plaques are Lp(a) based.

There used to be reliable Lp(a) measurements, but the FDA allowed the big blood testing companies to "compute" (rather than measure Lp(a)). A measured lp(a) test is a good indicator. You might try Life Extension.

Again, you can get an eye exam, and ask for the $9 retinal photo. Ask the optometrist what he thinks about your underlying cardiovascular disease, and see if he will circle the areas where he sees the white atheromas. (We used to send these photos to the late Sydney Bush, rest his sould), If the optometris or opthamologiset can't see any, that's good news too. We have posted medical journal articles that debated whether CVD drugs should be given prior to symptoms based on these photos showing narrowing and plaque. To most doctors, the idea that this condition can be reversed is not considered a possibility

[Lucky1]