Post
by ofonorow » Thu Jun 01, 2017 4:54 am
Not used to those units, but assume 1556mg/L is 155.6 mg/dl (dividing by 10). What is your total cholesterol, and especially LDL? (If your reported Lp(a) is HIGHER than LDL, there is a problem because Lp(a) is a SUBSET of LDL cholesterol. And in that case, my guess is that the Lp(a) is "calculated" (e.g. useless) rather than measured.
In any case YOUR ARE DOING EXACTLY THE RIGHT THING IF YOUR Lp(a) IS INDEED THAT HIGH. Pauling and Rath invented Lp(a) binding inhibitors, basically lysine (and proline) added to vitamin C. These orthomolecular substances at the proper dosages (you seem to be okay or close) will inactivate Lp(a) in the blood making it "less sticky." So the higher the Lp(a), the more you need Pauling's therapy. Capice?
As far as lowering Lp(a), there is little data/science to support such a phenomenon - other than reports at this forum. There is anecdotal evidence that the proline plays a pivotal role in lowering Lp(a). I know that johnwen's and my Lp(a) numbers are less than something like 3 mg/dl.
There is an interesting report from a Life Extension consultation. The LEF.ORG advisor found a study that carnitine lowers Lp(a).
Added - I thought I had read somewhere, maybe another post, how much vitamin C, lysine and proline you are taking. How much are you taking?
Owen R. Fonorow
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