This article/study pretty well describes formation of a plaque that is trying to heal the artery and then grows(due to lp(a) via LBS?)
-----"Working with specially bred Ldlr mice that were fed a Western diet, the team found that "platelets drive atherogenesis by skewing plaque macrophages into an inflammatory phenotype."
-->
>>the article does not explain WHY this happens or how to prevent if from occurring..... didnt pauling/rath talk about lack of collagen/vit C back in the Pauling figured this out when? at least 1993?
https://paulingblog.wordpress.com/tag/matthias-rath/Hardening of the arteries: Platelets, inflammation and a rogue protein conspire against the heart
The Lipid Hypothesis of coronary artery disease has long held that a high level of cholesterol is the causative factor of atherosclerosis, but a group of New York scientists is making a powerful case for platelets as the trigger of an artery-damaging inflammatory response.
Heart disease, a term that refers to a vast number of complex conditions, is most frequently used to define coronary artery disease, or atherosclerosis. The condition is a leading cause of cardiovascular deaths in Western countries and is marked by arterial deposits of plaque, which is composed of cholesterol, fatty material, calcium deposits and a motley collection of biological debris.
Over time, plaque not only hardens, but causes afflicted arteries to narrow, limiting the free flow of oxygen-rich blood. The Lipid Hypothesis, which continues to hold sway, is also known as the Cholesterol Hypothesis. It isn't, however, the entire story of how plaque forms and narrowed arteries set the stage for heart attack and stroke.
https://medicalxpress.com/news/2019-11- ... rogue.html