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Glucose is the protective biochemical critical to the brain because it places a veil of prtection over sensitive brain and neurological tissues. Medical research has not yet tapped into an understanding of just how much glucose the brain requires to function in times of stress --and just how critical it is that there's ample glucose reserved in the storage bank of the brain.
GLucose's protective veil is necessary for two reasons: First, glucose is needed to prevent brain cells, brain tissue, and neurons from becoming saturated by the acidic and corrosive nature of the adrenaline and cortisol rlease from anger....
Second, glucose is there to stop the electrical storms in the brain that arise when trauma occurs, with electrical impulses firing off at an alarming rate, ...
ofonorow wrote:Now that I no longer have diabetes, the amount of insulin I need has been reduced. Never had episodes of hypoglycemia eating fats.
Diabetes mellitus is characterized by recurrent or persistent high blood sugar, and is diagnosed by demonstrating any one of the following:[56]
Fasting plasma glucose level ? 7.0 mmol/l (126 mg/dl)
Plasma glucose ? 11.1 mmol/l (200 mg/dl) two hours after a 75 gram oral glucose load as in a glucose tolerance test (OGTT)
Symptoms of high blood sugar and casual plasma glucose ? 11.1 mmol/l (200 mg/dl)
Glycated hemoglobin (HbA1C) ? 48 mmol/mol (? 6.5 DCCT %).[67]
A positive result, in the absence of unequivocal high blood sugar, should be confirmed by a repeat of any of the above methods on a different day. It is preferable to measure a fasting glucose level because of the ease of measurement and the considerable time commitment of formal glucose tolerance testing, which takes two hours to complete and offers no prognostic advantage over the fasting test.[68] According to the current definition, two fasting glucose measurements above 7.0 mmol/l (126 mg/dl) is considered diagnostic for diabetes mellitus.
Per the WHO, people with fasting glucose levels from 6.1 to 6.9 mmol/l (110 to 125 mg/dl) are considered to have impaired fasting glucose.[69] people with plasma glucose at or above 7.8 mmol/l (140 mg/dl), but not over 11.1 mmol/l (200 mg/dl), two hours after a 75 gram oral glucose load are considered to have impaired glucose tolerance. Of these two prediabetic states, the latter in particular is a major risk factor for progression to full-blown diabetes mellitus, as well as cardiovascular disease.[70] The American Diabetes Association (ADA) since 2003 uses a slightly different range for impaired fasting glucose of 5.6 to 6.9 mmol/l (100 to 125 mg/dl).[71]
Glycated hemoglobin is better than fasting glucose for determining risks of cardiovascular disease and death from any cause.[72]
ofonorow wrote:I'm saying that I am not longer Type-II (although I never had insulin resistance)
My sugars before would go much higher and stay higher much longer.
ofonorow wrote:Since they have no idea what diabetes really is or what causes it, why should I care about THEIR definitions
And yes, I am insulin-dependent. So, if i were to take insulin with the meal, it would "look better. " Its a pain, so I take it after dinner, before bed.
Since I do not make "enough" insulin, it doesn't matter whether the "Type II" problem is completely resolved or not. By cured, I'm saying that nothing is blocking glucose from entering cells, as it was when i was eating fats.
My sugars before would go much higher and stay higher much longer.
And my diet, which now includes potatoes, pasta, bread, even my favorite candies, should be shooting my sugars through the roof. Instead, no real issues, unless I add some fat.. then I notice the sugars become less controllable.
ofonorow wrote:Now that I no longer have diabetes, the amount of insulin I need has been reduced. Never had episodes of hypoglycemia eating fats.
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