https://www.nature.com/articles/s41598-024-78859-5
We conclude that, by maintaining proteome stability and kinase-dependent signalling, both CBG and EAA may support the recovery of human keratinocytes exposed to UVB radiation, especially if applied together, while the time-dependence of these effects should be further studied.
Looking at this study in the perspective of Jack Kruse, UVB light is a driving force in our health, and not dangerous - so long as we don't wear sun glasses or sun screen. The non intuitive reason is that our eyes have sensors to detect UV light. When we block these sensors, our physiology changes and the light becomes more dangerous to our skin. When our bodies are "aware" that we are receiving UV, we won't sunburn.
But the idea of using vitamin C to protect the skin is always welcome, and it is very interesting that thy utilized an "ethylized" version of vitamin C in this study.